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Background The use of pulse field ablation in pulmonary vein isolation for treating atrial fibrillation is becoming more widespread. This method may create larger, more uniform, and deeper lesions compared to other techniques. Consequently, it could lead to the formation of a narrow conductive isthmus on the posterior wall, potentially heightening the likelihood of arrhythmias.Methods Electroanatomic mapping of the left atrium during coronary sinus pacing was performed before and after pulsefield ablation pulmonary vein isolation using the Carto electroanatomic mapping system and using a Pentaray mapping catheter. Electroanatomic maps were converted into an OpenEP format and analysed using EPWorkbench. The posterior wall conducting channel was measured as the distance between 0.5mV isolines atthree levels (roof, mid and inferior). Conducting channel width was defined as the average of these three measurements and also quantified as a proportion of the inter-vein distance. Conduction velocity within the posterior wall region was quantified pre- and post-ablation. (See image 1)Results Analysis was completed in 35 patients (77% male, mean age 60.8 years, range 24–80 years). As expected the conduction channel width was significantly reduced post pulse fi eld ablation with the channel width approximately 38% of the pre-ablation width. ( table 1)Conclusions Following pulse field ablation pulmonary vein isolation there is a wide range of conducting channel widths remaining on the posterior wall, with conduction properties of these channels unchanged from pre-ablation. Further research is needed to identify whether a specific minimum channel width impacts conduction properties and increases the likelihood of atrial tachycardia in patients following pulse field ablation.Abstract 83 Table 1Abstract 83 Image 1